An Individualized Dosing Regimen Of Ranibizumab 0.5 Mg Driven By Stabilization Criteria For Up To 12 Months Resulted In Significant Best-corrected Visual Acuity Gain In A Broad Population Of Patients With Macular Enema Secondary To Crvo, Including Those With Macular Ischemia At Baseline.

An individualized dosing regimen of ranibizumab 0.5 mg driven by stabilization criteria for up to 12 months resulted in significant best-corrected visual acuity gain in a broad population of patients with macular enema secondary to CRVO, including those with macular ischemia at baseline. This is called an occlusion. Therefore, we must at first know which type of CRVO we are dealing with, and that makes differentiation of two types of CRVO the first essential step in the management of CRVO. Retinal artery occlusion is caused by blood clots, fat, or plaque that gets caught in the arteries. Visual loss may have been preceded by transient loss of vision in the past amaurosis fugal in the case of embolic sources. The condition occurs most often in men and women over the age of 50, particularly those in their 60s and 70s.  The most severe is occlusion of the ophthalmic artery. The retinal veins drain away the used blood from the retinal cells. -Angiogram shows no filling of arteries retina is oenematous and pallid, Cherry red = seeing through to choroid al circulation ciao- pallid and cherry red spot perfusion due to a cilioretinal artery branch of biliary circulation not retinal circulation majority: Embolus at lamina cribrosa = Calcific Cardiac origin or Fibrin Platelet or Cholesterol Carotid origin May have 20/30 or 20/40 vision but can't see loss of peripheral VF CRAO-Pallid, enema, hypo perfusion, multiple emboli, cilioretinal artery does not seem well perfused Ono categories in CRAO all ischemic! FDA approves Lucentis ranibizumab injection for the treatment of macular edema following retinal vein occlusion.

Central vein occlusion study of photo coagulation therapy. Twenty-five 75.8% of the 33 patients had follow-up examinations for a mean period of 9.1 months. Once the retinopathy burns itself out, the stimulus for neovascularization disappears and consequently the anterior segment neovascularization spontaneously starts to regress - a fact usually not appreciated in the management of these eyes. Most of the time, this medical treatment regimen is enough to keep the intra ocular pressure under satisfactory control. For retinal detachments, laser surgery is used to fuse the retina to its underlying layer.

Retinal vascular occlusion